N-acetylcysteine alleviates phenylephrine-induced cardiomyocyte dysfunction via engaging PI3K/AKT signaling pathway.

Sheng-Ping Chao,Wen-Lin Cheng,Wenjuan Yi,Huan-Huan Cai,Keqiong Deng,Jian-Lei Cao,Ziyue Zeng,Hairong Wang,Xiaoyan Wu

American journal of hypertension（2023）

引用 0|浏览6

暂无评分

摘要

Collected, NAC could attenuate cardiomyocyte dysfunction subjected to phenylephrine, partially by regulating the ROS-induced PI3K/AKT dependent signaling pathway.

查看译文

关键词

apoptosis, blood pressure, cardiomyocytes, hypertension, hypertrophy, N-acetylcysteine, oxidative stress, PI3K/AKT

AI 理解论文

溯源树

样例

生成溯源树，研究论文发展脉络

Chat Paper

正在生成论文摘要