Inhibition of NK1.1 signaling attenuates pressure overload-induced heart failure, and consequent pulmonary inflammation and remodeling.

Xiaochen He, Rui Xu,Lihong Pan,Umesh Bhattarai,Xiaoguang Liu,Heng Zeng,Jian-Xiong Chen,Michael E Hall,Yingjie Chen

Frontiers in immunology（2023）

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摘要

Our data suggest that inhibition of NK1.1 signaling is effective in attenuating systolic overload-induced cardiac fibrosis, dysfunction, and consequent pulmonary remodeling in immunocompetent mice through modulating the cardiopulmonary inflammatory response.

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关键词

consequent pulmonary inflammation,heart failure,overload-induced

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