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Chronic alcohol consumption shifts learning strategies and synaptic plasticity from hippocampus to striatum-dependent pathways.

Léa Tochon,Rose-Marie Vouimba,Marc Corio,Nadia Henkous,Daniel Béracochéa,Jean-Louis Guillou,Vincent David

Frontiers in psychiatry（2023）

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Abstract
These results support the view that CAC disrupt normal hippocampo-striatal interactions, and suggest that targeting this cognitive imbalance through spatial/declarative task training could be of great help to maintain protracted abstinence in alcoholic patients.
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Key words
alcohol, learning strategies, memory systems, hippocampus (CA1), dorsal striatum, synaptic plasticity, addiction, amygdala
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