Developmental and cardiac toxicity assessment of Ethyl 3-(N-butylacetamido) propanoate (EBAAP) in zebrafish embryos

•BAAPE induced developmental defects in zebrafish embryos at early stages.•Embryos exposure to BAAPE with a lethal concentration 50 (LC50) of 140 mg/L at 72 hours post fertilization (hpf).•BAAPE caused cardiotoxicity and changed the expression levels of key genes in cardiac development in zebrafish larvae.•BAAPE exposure induced production and accumulation of ROS and enhanced oxidative stress levels.•BAAPE caused mitochondria-mediated apoptosis in zebrafish larvae.