Failure to decrease HbA1c levels following TB treatment is associated with elevated Th1/Th17 CD4+ responses.

Robert Krause,Christian M Warren,Joshua D Simmons,Peter F Rebeiro,Fernanda Maruri,Farina Karim,Timothy R Sterling,John R Koethe,Al Leslie,Yuri F van der Heijden

Frontiers in immunology（2023）

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摘要

Overall, these data suggest that patients with stable/increased HbA1c had an increased pro-inflammatory state. Persistent inflammation and elevated T cell activity in individuals with unresolved dysglycemia following TB treatment may indicate failure to fully resolve infection or may promote persistent dysglycemia in these individuals, and further studies are needed to explore potential mechanisms.

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关键词

hyperglycemia,tuberculosis,diabetes,HIV,HbA1c,IL-17,TNF-alpha,CX3CR1

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