Supplementary Table 2 from CBP Loss Cooperates with PTEN Haploinsufficiency to Drive Prostate Cancer: Implications for Epigenetic Therapy

Liya Ding,Shuai Chen,Ping Liu,Yunqian Pan,Jian Zhong,Kevin M. Regan,Liguo Wang,Chunrong Yu,Anthony Rizzardi, Liang Cheng,Jun Zhang,Stephen C. Schmechel,John C. Cheville,Jan Van Deursen,Donald J. Tindall,Haojie Huang

crossref（2023）

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Abstract

PDF file - 66KB, Supplemental Table S2. The incidence of prostatic intraepithelial neoplasia (PIN) lesions detected in prostate-specific Cbp homozygous and Pten heterozygous deletion (Cbppc-/-;Ptenpc+/-) mice at 4 and 6 months of age and their littermate controls, including Cbp homozygous deletion alone (Cbppc-/-), Pten heterozygous deletion alone (Ptenpc+/-) and 'wild type' loxp (CbpL/L;PtenL/+) mice.

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