TU-100 Antagonizes the M2 Polarization Phenotype of Macrophages in the Tumor Microenvironment by Suppressing the TLR4/NF-B/STAT3 Axis.

Shuhai Chen,Yuji Morine,Y U Saito,Shinichiro Yamada,Hiroki Teraoku,Tetsuya Ikemoto,Mitsuo Shimada

Anticancer research（2023）

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摘要

TU-100 may alleviate the progression of cancer by regulating the M2 polarization of macrophages within the TME, suggesting a viable therapeutic approach.

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关键词

M2 polarization,Toll-like receptor 4,Tumor-associated macrophages,nuclear factor-B,tumor microenvironment

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