Supplementary Figures 1-6 from SLX4IP Promotes Telomere Maintenance in Androgen Receptor–Independent Castration-Resistant Prostate Cancer through ALT-like Telomeric PML Localization

S1: Validation of TMM characterization techniques. S2: Expression of SLX4IP in mCRPC versus primary disease. S3: SLX4IP knockdown correlates with accelerated telomere shortening. S4: SLX4IP knockdown is accompanied by a blunted atypical ALT phenotype. S5: SLX4IP knockdown promotes telomere shortening and senescence. S6: Androgen-independent marker expression and telomere length changes following androgen deprivation.