Hepatic follistatin increases basal metabolic rate and attenuates diet-induced obesity during hepatic insulin resistance.
Molecular metabolism（2023）
Abstract
Thus, complete hepatic insulin resistance in LDKO mice fed a HFD revealed Fst-mediated communication between the liver and muscle, which might go unnoticed during ordinary hepatic insulin resistance as a mechanism to increase muscle EE and constrain obesity.
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Key words
Energy expenditure,Follistatin,FoxO1,Insulin receptor substrate,Insulin resistance,Obesity
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