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Neutrophils Protect Against Staphylococcus aureus Endocarditis Progression Independent of Extracellular Trap Release.

Severien Meyers,Marleen Lox,Sirima Kraisin,Laurens Liesenborghs,Caroline P Martens,Liesbeth Frederix,Stijn Van Bruggen,Marilena Crescente,Dominique Missiakas,Pieter Baatsen,Thomas Vanassche,Peter Verhamme,Kimberly Martinod

Arteriosclerosis, thrombosis, and vascular biology（2023）

Cited 2|Views20
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Abstract
Neutrophils protect against IE independent of NET release. Absence of coagulases, but not nucleases, reduced IE severity and increased NET levels. Staphylocoagulase-induced fibrin likely hampers NETs from constraining infection and the resultant tissue damage, a hallmark of valve destruction in IE.
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Key words
Staphylococcus aureus,coagulases,endocarditis,extracellular traps,neutrophils
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