Overexpression of lipocalin 2 in PBX1-deficient decidual NK cells promotes inflammation at the maternal-fetal interface.
American journal of reproductive immunology (New York, N.Y. : 1989)(2023)
摘要
Excessive LCN2 secreted by PBX1 dNK cells at the maternal-fetal interface recruit neutrophils and causes an inflammatory response, which is related to FGR.
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关键词
PBX1,decidual natural killer cells,inflammation LCN2,maternal-fetal interface
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