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EGR1/NOX4 pathway regulates oxidative stress and further facilitates fibrosis progression in keloids responses to TGF-β1

Haiyan Qin,Lianbo Zhang,Mingxi Li,Ying Liu,Shilong Sun,Wenting Nie,Bing Bai,Gaokai Li,Guang Zhang

Journal of Dermatological Science（2022）

Cited 5|Views1
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Abstract
Our findings provide new insights into the mechanisms of the TGF-β1/EGR1/NOX4 pathway in keloid fibrosis, and the TGF-β1/EGR1/NOX4 axis may serve as a potential therapeutic target for keloids.
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Key words
EGR1,Fibrosis,Keloid,NOX4,Oxidative stress
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