Non-canonical Small GTPase RBJ Promotes NSCLC Progression Through the Canonical MEK/ERK Signaling Pathway.

Yujin Wang,Xiaoyan Shen,Qingwen Wang,Zixin Guo,Liwen Hu,Zhe Dong,Weidong Hu

Current pharmaceutical design（2022）

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摘要

RBJ promoted cancer progression in NSCLC by activating EMT via the MEK/ERK signaling. Thus, RBJ could be used as a potential therapeutic against NSCLC.

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关键词

GTPase,MEK/ERK,NSCLC,RBJ,Ras superfamily,epithelial-mesenchymal transition (EMT)

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