Smoking Habits Do Not Affect Biological Responses Induced by Leucocyte and Platelet‐Rich Fibrin in periodontal ligament cells

Leucocyte- and platelet-rich fibrin derived from smokers could be an autologous source of biomolecules to stimulate cell biological activities involved in wound healing in smokers who have difficulties in ceasing this habit. Clinical trials are required to evaluate the impact of leucocyte- and platelet-rich fibrin on healing responses in smokers.