UPREGULATION OF NADH/NADPH OXIDASE 4 BY ANGIOTENSIN II INDUCES PODOCYTE APOPTOSIS
NEPHROLOGY DIALYSIS TRANSPLANTATION（2022）
摘要
These findings suggest that angiotensin II promoted podocyte mitochondrial oxidative stress resulting in apoptosis via the upregulation of NADH/NADPH oxidase 4 and angiotensin II type 1 receptor and, which could be prevented by NADH/NADPH oxidase 4 inhibition and/or antagonizing angiotensin II type 1 receptor as well as antioxidants.
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