SNX27-mediated endocytic recycling of GLUT1 is suppressed by SARS-CoV-2 spike, possibly explaining neuromuscular disorders in patients with COVID-19

Yongwen Ren,Yihan Liu,Zhiwen Zhang,Yuwen Liu,Kangxin Li,Leiliang Zhang

JOURNAL OF INFECTION（2022）

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摘要

•SARS-CoV-2 S inhibits the interaction between SNX27 and its cargo GLUT1.•SARS-CoV-2 S disrupts the interaction between SNX27 and retromer component Vps26A.•Endocytic recycling of GLUT1 mediated by SNX27 is suppressed by SARS-CoV-2 S.•GLUT1 deficiency by S could explain neuromuscular disorders in long COVID patients.

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