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The Potential Mechanism Behind Native and Therapeutic Collaterals in Moyamoya.

Xiang-Yang Bao,Yan-Na Fan,Qian-Nan Wang,Xiao-Peng Wang,Ri-Miao Yang,Zheng-Xing Zou,Qian Zhang,De-Sheng Li,Lian Duan,Xin-Guang Yu

Frontiers in neurology（2022）

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Abstract
These data imply that mutations of RNF213 p.R4810K affect the establishment of spontaneous collateral circulation, and EPCs are involved in the process of formation of new EDAS collaterals.
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Key words
RNF213 p.R4810K,clinical phenotype,collaterals,moyamoya disease,mutation
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