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Interleukin‐19 Ameliorates Experimental Autoimmune Encephalitis

Hideyuki Takeuchi, Hiroshi Horiuchi, Bijay Parajuli, Hiroyasu Komiya, Fumiaki Tanaka, Akio Suzumura

Clinical & experimental neuroimmunology（2021）

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Abstract
Interleukin-19 serves as a negative feedback regulator to limit the pro-inflammatory response of macrophages and microglia in autocrine/paracrine manners. Interleukin-19 suppresses microglia/macrophage antigen presentation, T helper 17 cell expansion and subsequent inflammation in the central nervous system, resulting in improvement in experimental autoimmune encephalomyelitis.
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