Significant Role Of Cellular Activation In Viral Infections

Immune response during viral infection is the result of pathogen recognition and antibody production by the memory cells residing in the host target tissue. These cells by origin are differentiated from the cytotoxic CD8(+) T cells and play a major role in developing adaptive immunity. Early activation of these T cells by ribosomal binding proteins during various cell death pathways can induce a critical autoimmune response suitable to develop enhanced strategic immune therapies for many chronic viral infections. In addition, there are various genes which get expressed in good amount in the process of T-cell activation during infections like Epstein-Barr virus. Focusing on these changes can help in determining the pathogenic process and orientation of the viral infection. Since we can only control but not cure most of the viral infections using present medications, following these novel strategies can help in focusing on the host cell death pathways, thereby developing the drugs that can give complete immunity against virus invasion by facilitated viral cell elimination from the humoral environment.