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Loss of KMT2C reprograms the epigenomic landscape in hPSCs resulting in NODAL overexpression and a failure of hemogenic endothelium specification

Shailendra Maurya,Wei Yang,Minori Tamai,Qiang Zhang,Petra Erdmann-Gilmore,Amelia Bystry,Fernanda Martins Rodrigues,Mark C. Valentine,Wing H. Wong,Reid Townsend,Todd E. Druley

EPIGENETICS（2022）

Cited 6|Views38
Key words
Histone methyltransferases,development,gene expression,chromatin remodelling,hemogenic endothelium,pluripotency,mesoderm
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