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S100A7表达下调阻断NF-κB途径诱导胃癌SGC-7901细胞凋亡

赵洁,郭长升,韩怀忠,徐琦,宋书杰,孙立东,石磊,常勤征,盛之玲

Academic Journal of Second Military　Medical University（2013）

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Abstract
S100A7与肿瘤的发生、发展密切相关.我们的前期研究显示,胃癌组织和细胞中均呈现S100A7的过表达[1-2],并且其表达下调能明显抑制胃癌SGC-7901细胞的增殖和迁移[2].本研究中我们通过流式细胞术分析S100A7表达下调对胃癌SGC-7901细胞凋亡的影响,并探讨其与NF-κB途径之间的关系,为以S100A7为靶点的胃癌基因治疗提供理论依据. 1 材料和方法 1.1 实验材料 胃癌细胞株SGC-7901购自中国科学院上海细胞研究所.
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