ERK途径参与未受精蚕卵的孤雌激活
Bulletin of Sericulture（2017）
Abstract
U0126是激酶MEK1/2的特异性抑制剂,可阻止下游MAPK激酶磷酸化,导致减数分裂阻滞机构失活,卵细胞恢复分裂.本实验以抑制剂U0126注射羽化前2天的雌蛹,羽化后孤雌生殖诱导未受精卵,并调查蚕卵的发育情况.结果表明:对孤雌生殖发生率不高的Nastari品系,U0126可极显著提高孤雌生殖发生率;对孤雌生殖发生率高的品系无14,U0126可部分提高孤雌生殖发生率,暗示ERK途径参与蚕卵孤雌激活.
MoreAI Read Science
Must-Reading Tree
Example
Generate MRT to find the research sequence of this paper
Chat Paper
Summary is being generated by the instructions you defined