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Mitomycin C Induces Pulmonary Vascular Endothelial-to-mesenchymal Transition and Pulmonary Veno-occlusive Disease via Smad3-dependent Pathway in Rats.

Chenting Zhang,Wenju Lu,Xiaoyun Luo,Shiyun Liu,Yi Li,Qiuyu Zheng,Wenyan Liu,Xuefen Wu,Yuqin Chen, Qian Jiang,Zizhou Zhang,Guoping Gu,Jiyuan Chen,Haixia Chen,Jing Liao,Chunli Liu,Cheng Hong,Haiyang Tang,Dejun Sun,Kai Yang,Jian Wang

AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE（2020）

Cited 8|Views23
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Abstract
Our data indicated that targeted inhibition of Smad3 leads to a potential, novel strategy for PVOD therapy, likely by inhibiting the EndoMT in pulmonary microvasculature.
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Key words
Pulmonary veno-occlusive disease,Smad3,endothelial-to-mesenchymal transition,mitomycin C,pulmonary microvascular endothelial cell
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