Early adolescent Rai 1 reactivation reverses transcriptional and social interaction deficits in a mouse model of Smith – Magenis syndrome

semanticscholar(2018)

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摘要
Howard Hughes Medical Institute, Stanford University, Stanford, CA 94305; Department of Biology, Stanford University, Stanford, CA 94305; Neurosciences Program, Stanford University, Stanford, CA 94305; Stanford Behavioral and Functional Neuroscience Laboratory, Stanford University School of Medicine, Stanford, CA 94305; Interdisciplinary Institute of Neuroscience and Technology, Zhejiang University, 310012 Hangzhou, People’s Republic of China; Qiushi Academy for Advanced Studies, Zhejiang University, 310012 Hangzhou, People’s Republic of China; and Department of Neurosurgery, Stanford University School of Medicine, Stanford, CA 94305
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