3 Use of Antibiotics and Subsequent Risk of Systemic Lupus Erythematosus : A Matched Case-Control Study

J. T. Merrill, M. E. Alarcón-Riquelme, M. Petri, E. R. Dow, E. Nantz, L. K. Nisenbaum, K. M. Schroeder, W. J. Komocsar, N. B. Perumal, G. V. Rocha, R. E. Higgs, C. Aranow, G. Bertsias, E. Silva Dutra de Oliviera, Bonfa, R. Cervera, N. Costedoat-Chalumeau, T. Dörner, F. Houssiau, K., Lerstrom, E. Morand, M. Mosca, S. Navarra, M. Urowitz, A., Voskuijl, A. Voss, M. Ward, V. Werth

semanticscholar（2015）

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Abstract

mote both the production and response to type I IFN. There is also a lack of proper regulation in SLE of cells in the type I IFN system and recently, we observed that autoantibodies to NK cells may contribute to the activation of the type I IFN system. These NK cells autoantibodies block the inhibitory NK cells receptors and were found in an SLE subset with an active and severe disease phenotype.

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