Galectin-3: A Cardiomyocyte Antiapoptotic Mediator at 24-Hour Post Myocardial Infarction.
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology（2020）
Abstract
We conclude that the increased levels of GAL-3 at 24-hour following MI regulate antiapoptotic mechanisms in the myocardium that will shape the future course of the disease. We also identified that the anti-apoptotic mechanisms are likely mediated through interaction of GAL-3 with Akt-1, NF kappa-B, beta- catenin and cathepsin D proteins.
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Key words
Heart, Myocardial infarction, Galectin 3, Apoptosis
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