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Early life tobacco exposure and children’s telomere length: the HELIX Project

Citlalli Osorio-Yáñez,Diana B.P. Clemente,Lea Maitre,Martha Vives-Usano,Mariona Bustamante,David Martinez,Maribel Casas,Jan Alexander,Cathrine Thomsen,Leda Chatzi,Kristine B. Gützkow,Regina Grazuleviciene,Dries S. Martens,Michelle Plusquin,Remy Slama,Rosemary Rc McEachan,John Wright,Tiffany C Yang,Jose Urquiza,Ibon Tamayo,Jordi Sunyer,Marina Vefeiadi,Tim S. Nawrot,Martine Vrijheid

Science of The Total Environment（2020）

Cited 16|Views86
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Abstract
•Studies on prenatal exposure to tobacco and children's LTL and mtDNA are limited.•Maternal cotinine during pregnancy is associated with shorter LTL in children.•Global tobacco exposure is linked to mitochondrial DNA increase in children.•Maternal smoking during pregnancy may induce biological aging from an early age.
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Key words
Tobacco exposure,Leukocyte telomere length,Mitochondrial DNA content,Pregnancy,HELIX
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