Neonatal Rat Ventriculomyocytes are Stimulated to Undergo Apoptosis through a β1-Selective Adrenergic Pathway

Conference Abstract| February 01 2001 Neonatal Rat Ventriculomyocytes are Stimulated to Undergo Apoptosis through a β1-Selective Adrenergic Pathway RS Foo; RS Foo 1Clinical Pharmacology Unit and Division of Cardiovascular Medicine, Addenbrooke's Hospital, Cambridge CB2 2QQ, England Search for other works by this author on: This Site PubMed Google Scholar PE Glennon; PE Glennon 1Clinical Pharmacology Unit and Division of Cardiovascular Medicine, Addenbrooke's Hospital, Cambridge CB2 2QQ, England Search for other works by this author on: This Site PubMed Google Scholar MR Bennett; MR Bennett 1Clinical Pharmacology Unit and Division of Cardiovascular Medicine, Addenbrooke's Hospital, Cambridge CB2 2QQ, England Search for other works by this author on: This Site PubMed Google Scholar MJ Brown MJ Brown 1Clinical Pharmacology Unit and Division of Cardiovascular Medicine, Addenbrooke's Hospital, Cambridge CB2 2QQ, England Search for other works by this author on: This Site PubMed Google Scholar Clin Sci (Lond) (2001) 100 (s44): 1P. https://doi.org/10.1042/cs100001P Views Icon Views Article contents Figures & tables Video Audio Supplementary Data Peer Review Share Icon Share Twitter LinkedIn Cite Icon Cite Get Permissions Citation RS Foo, PE Glennon, MR Bennett, MJ Brown; Neonatal Rat Ventriculomyocytes are Stimulated to Undergo Apoptosis through a β1-Selective Adrenergic Pathway. Clin Sci (Lond) 1 February 2001; 100 (s44): 1P. doi: https://doi.org/10.1042/cs100001P Download citation file: Ris (Zotero) Reference Manager EasyBib Bookends Mendeley Papers EndNote RefWorks BibTex toolbar search Search Dropdown Menu nav search search input Search input auto suggest search filter All ContentAll JournalsClinical Science Search Advanced Search This content is only available as a PDF. © 2001 The Biochemical Society and the Medical Research Society2001 Article PDF first page preview Close Modal You do not currently have access to this content.