脑缺血再灌注损伤中IL-1β的作用及机制

Journal of Beihua University(2004)

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Abstract
生理情况下,白细胞介素-1β在脑内表达很低,但脑缺血再灌注后其表达大量增加,其机制与诱导粘附分子合成,激活内皮细胞产生多种活性因子,促进自由基的产生,增加细胞内的Ca2+浓度和促进NO的合成有关.
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Neuroprotective Effects
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