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Kupffer cells activation promoted binge drinking-induced fatty liver by activating lipolysis in white adipose tissues

Yu-Ying Zhao,Rui Yang,Mo Xiao,Min-Jie Guan,Ning Zhao,Tao Zeng

Toxicology（2017）

Cited 16|Views5
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Abstract
•GdCl3 and etanercept attenuated binge drinking-induced hepatic TG accumulation by a similar amount.•GdCl3 and etanercept had no effects on ethanol-induced disturbance of PPAR-α and SREBP-1c.•GdCl3 and etanercept suppressed binge drinking-induced phosphorylation of HSL.•TNF-α promoted HSL phosphorylation and lipolysis in cultured epididymal adipose tissues.
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ACC,ACOX,AFL,ALD,ALT,AST,AMPK,CPT-1,CYP2E1,FAS,FFA,HSL,KCs,LFABP,LPS,MDA,NO,PPAR-α,ROS,SREBP-1c,TG,TNF-α
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