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Polygenic burdens on cell-specific pathways underlie the risk of rheumatoid arthritis

Kazuyoshi Ishigaki,Yuta Kochi,Akari Suzuki,Yumi Tsuchida,Haruka Tsuchiya,Shuji Sumitomo,Kensuke Yamaguchi,Yasuo Nagafuchi,Shinichiro Nakachi,Rika Kato,Keiichi Sakurai,Hirofumi Shoda,Katsunori Ikari,Atsuo Taniguchi,Hisashi Yamanaka,Fuyuki Miya,Tatsuhiko Tsunoda,Yukinori Okada,Yukihide Momozawa,Yoichiro Kamatani,Ryo Yamada,Michiaki Kubo,Keishi Fujio,Kazuhiko Yamamoto

NATURE GENETICS（2017）

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Abstract
Yuta Kochi and colleagues perform expression quantitative trait loci (eQTL) analysis on five subsets of immune cells individually sorted from blood from 105 individuals. They develop an integrated analysis pipeline of expression and epigenomic data along with gene association to identify cell-specific candidate causal genes and apply this to rheumatoid arthritis.
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Key words
Gene regulation,Rheumatoid arthritis,Biomedicine,general,Human Genetics,Cancer Research,Agriculture,Gene Function,Animal Genetics and Genomics
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