Endoplasmic reticulum stress induced by tunicamycin increases resistin messenger ribonucleic acid through the pancreatic endoplasmic reticulum eukaryotic initiation factor 2α kinase-activating transcription factor 4-CAAT/enhancer binding protein-α homologous protein pathway in THP-1 human monocytes.

Junpei Hamada,Hiroshi Onuma,Fumihiro Ochi,Hiroki Hirai,Koji Takemoto, Akiko Miyoshi, Manami Matsushita, Yuko Kadota,Jun Ohashi,Ryoichi Kawamura,Yasunori Takata,Wataru Nishida,Seiichi Hashida,Eiichi Ishii,Haruhiko Osawa

JOURNAL OF DIABETES INVESTIGATION（2016）

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摘要

Endoplasmic reticulum stress induced by tunicamycin increased resistin mRNA through the PERK-ATF4-CHOP pathway in THP-1 human monocytes. ER stress could lead to insulin resistance through enhanced resistin gene expression in human monocytes.

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关键词

Endoplasmic reticulum stress,Human monocytes,Resistin

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