STAT3 in arsenic lung carcinogenicity.
Oncoimmunology（2015）
Abstract
We recently found that the chronic sterile inflammation contributes to arsenic lung tumorigenesis which is inhibited by autophagy. regulates the interaction between inflammation and autophagy. may also play a critical role in mediating the crosstalk between lung epithelial cells and their microenvironment, including immune cells, during arsenic lung carcinogenesis.
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Key words
STAT3,arsenic,autophagy,immunosurveillance,lung tumorigenesis,microenvironment
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