异丙酚对大鼠心肌缺血再灌注损伤时细胞间黏附分子-1及核转录因子-κB表达的影响
SHANXI MEDICAL JOURNAL（2009）
Abstract
现已证实,缺血再灌注时血管内皮细胞核转录因子(NF)-κB活化可使细胞间黏附分子-1 (ICAM-1)表达上调[1].NF-κB的激活可能是一些炎症介质释放的关键因素,而ICAM-1与NF-κB之间以及诸多细胞因子间复杂的相互作用可使炎症信号进一步扩大和增强.
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