Response to Comment on "Thiamine deficiency promotes T cell infiltration in experimental autoimmune encephalomyelitis: the involvement of CCL2".

Zhe Ji et al. ([1][1]) reported that experimental autoimmune encephalomyelitis (induced either by MOG peptide immunization or by MOG-immune T cell transfer) was more severe in mice with thiamine deficiency (TD) than in mice without TD, and concluded that TD influences neuroinflammation by