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神经节苷脂对体外培养脊髓神经元的保护作用

王洪德,黄飞,孔祥梅,李泽福

Chinese Journal of Coal Industry Medicine（2006）

Cited 1|Views21
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Abstract
谷氨酸可通过兴奋性毒性和氧化性毒性二种途径对培养的神经元产生毒性作用.高浓度谷氨酸通过激活谷氨酸受体引起一系列病理变化,最终导致神经元肿胀、代谢紊乱而死亡.神经节苷脂(monosialoganglioside,GM1)能促进中枢神经元损伤后的恢复,并能抑制神经元的凋亡.
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