基本信息
浏览量:34
职业迁徙
个人简介
Research Divisions: Endocrine and Diabetes Platform
Expertise
Using tracer methodology to measure glucose turnover out of steady-state. The role of hormones and metabolites to regulate metabolic fluxes in physiology and diabetes.
Extra-pancreatic glucagon.
Research Interests:
The mechanisms of exercise and stress on the prevention of metabolic syndrome and development of type 2 diabetes. Role of excessive cortisol release in diabetes.
Effect of antecedent stress and hypoglycemia on the responses of the hypothalamic-pituitary-adrenal axis (gene expression of brain stress hormones and their receptors), and counterregulation (counterregulatory hormones, glucose turnover, adrenal medullary enzymes) in models of type 1 diabetes.
Molecular mechanisms responsible for inadequate response of counterregulatory hormones during insulin-induced hypoglycemia.
Prevention of hypoglycemia in type 1 diabetics, using somatostatin receptor antagonists. Improvement of deficient glucagon, cortisol, and catecholamine responses.
Physiology and pathophysiology of pancreatic α-cell in type 1 and type 2 diabetes.
Expertise
Using tracer methodology to measure glucose turnover out of steady-state. The role of hormones and metabolites to regulate metabolic fluxes in physiology and diabetes.
Extra-pancreatic glucagon.
Research Interests:
The mechanisms of exercise and stress on the prevention of metabolic syndrome and development of type 2 diabetes. Role of excessive cortisol release in diabetes.
Effect of antecedent stress and hypoglycemia on the responses of the hypothalamic-pituitary-adrenal axis (gene expression of brain stress hormones and their receptors), and counterregulation (counterregulatory hormones, glucose turnover, adrenal medullary enzymes) in models of type 1 diabetes.
Molecular mechanisms responsible for inadequate response of counterregulatory hormones during insulin-induced hypoglycemia.
Prevention of hypoglycemia in type 1 diabetics, using somatostatin receptor antagonists. Improvement of deficient glucagon, cortisol, and catecholamine responses.
Physiology and pathophysiology of pancreatic α-cell in type 1 and type 2 diabetes.
研究兴趣
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mag(2015)
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Erwan Leclair,Ashley Peckett, Emily Dunford, Trevor Teich, Rich-Ard Liggins, David Coy,Mladen Vranic,Michael C. Riddell
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