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Hannah Kinney's research is directed at defining the causes of sudden infant death syndrome (SIDS). Dr. Kinney and colleagues are testing the idea that SIDS, or a subset of SIDS, is due to a developmental brainstem defect in autonomic and/or respiratory control during sleep. Focusing specifically on the arcuate nucleus in the ventral medulla area of the brainstem -- important in the detection of carbon dioxide and other respiratory and blood pressure responses -- her team is identifying abnormalities that put an infant at risk for sudden death during sleep. While continuing to study the anatomy and neurochemistry of the ventral medulla in SIDS victims, Dr. Kinney's team is also looking at the function and pathology of the ventral medulla in animal models. The ultimate goals of this research are to define ventral medullary abnormalities in living infants and to suggest ways of preventing the abnormalities from leading to sudden infant death. Her studies have also detected serotonergic binding deficiencies in SIDS victims in six functionally and developmentally related components of ventral medulla--all regions critically involved in chemoreception, respiratory drive, blood pressure responses, upper airway reflexes, and/or thermoregulation.
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Brian L Edlow, Mark Olchanyi, Holly J Freeman,Jian Li,Chiara Maffei,Samuel B Snider,Lilla Zöllei,J Eugenio Iglesias,Jean Augustinack,Yelena G Bodien,Robin L Haynes,Douglas N Greve,
bioRxiv : the preprint server for biology (2023)
Arunnjah Vivekanandarajah,Morgan E Nelson,Hannah C Kinney,Amy J Elliott,Rebecca D Folkerth,Hoa Tran, Jacob Cotton, Perri Jacobs,Megan Minter,Kristin McMillan,Jhodie R Duncan,Kevin G Broadbelt,
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